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Sunday, December 6, 2009

Sex After Herpes - Yes You Can Still Have a Fulfilling Sex Life If You Have Genital Herpes

When you first get diagnosed with herpes, it can feel like the end of the world. You may feel like life as you know it is over, and one of the most common misconceptions about genital herpes is that you'll never have a normal sex life again. Well, I'm here to tell you that simply isn't true. With a few precautions and a lot of common sense, you'll find that you can have sex after herpes, and a varied and fulfilling sex life at that.

The first thing you have to realise is things are going to be a bit different from now on, and you are probably going to have to be a bit more careful than you were in the past. The first thing to know is you'll absolutely need to avoid penetrative sex if you or your partner is currently experiencing a herpes outbreak on your genitals. This is the main unfortunate downside to sex after herpes.

If both you and your partner between outbreaks, you can have full penetrative vaginal, anal or oral sex with very little risk, provided the herpes sufferer is on suppressive antivirals, and you use adequate protection such as a condom or dental dam.

And don't forget, there are a myriad of ways to explore lovemaking if you or your partner are having an outbreak of sores. You can try mutual masturbation, using toys or if the partner with the outbreak has doesn't suffer from oral herpes as well, he or she can safely perform oral sex on the non-infected partner.

Just be sure to always use protection and keep up your daily medication to help keep the risk of shedding the virus down. If you do have some intimacy during an outbreak, make sure you both wash your hands and body thoroughly afterwards. Sex after herpes might be a little different, but with a bit of care and some variety and imagination, you can still experience a satisfying and fulfilling sex life.

Visit Fitter Healthier today to read our free guide on herpes home treatments and check out our reviews of the most effective genital herpes treatments available today.

Stop putting up with the pain and discomfort of genital herpes today! Stop genital herpes dead in its tracks with our tried and tested natural treatments.

Article Source: http://EzineArticles.com/?expert=Kat_Samson

Friday, December 4, 2009

What Kind of Gout Medicine Can Relieve Gout Pain?

Gout pain can be very debilitating and can often restrict your movement. How do you get rid of this pain or what kind of gout medicine should you take to get some relief? Here is a list of gout medicines to take so that you can get relief from gout pain.

A popular gout medicine used to treat gout patients is colchicines, this medicine works by restricting the function of white blood cells, they can't move from one site to another. The patient is recommended 0.6 mg tablet every two hours till the pain subsides. Stop the medicine if you start feeling nauseous or have diarrhea.

This medicine also functions as a prophylactic drug, in other words it acts as a preventive medicine. During the initial six months of treatment when the medicine will work to reduce the uric acid level in blood, the patient may strangely have more attacks. After six months the patient can discontinue the use of colchicines.

The side effects of colchicines are also numerous so cautions should be observed while using it. Overuse can lead to kidney failure, calcium deficiency, heart problems, bone marrow suppression, lung failure, liver problems, rashes and even death.

How an individual's body responds to a particular medicine differs, often one gout medicine may completely work for one person while for the other there might be endless problems. When a patient takes cyclosporine along with colchicines there is likelihood of there being damage to the kidneys.

Patients who take colchicines on a regular basis should be monitored at regular intervals. This should include RBC count, liver and kidney function test and urine analysis.

Another drug that is commonly used in the treatment of gout is probenecid; it is a drug that helps in the excretion of excessive uric acid through urine. It blocks the process of the absorption of uric acid by kidney. This drug should be administered when the patient's health is stable and not when he is undergoing an attack.

The dosage begins with 500 mg once a day and later becomes 500 mgs twice a day. To ensure that probenecid is effective the kidney should function normally. The patient should also ensure good flow of urine by constant flow of urine.

Patients who suffer from kidney stones should not use probenecid.

Losartan a medicine for blood pressure also lowers uric acid levels in blood.

The above mentioned drugs should not be taken without consulting a medical practitioner so he/she can find what gout medicine is right for you.

Get gout relief today by learning more about gout medicine and what you can do to cure your gout.

Discover other alternatives to prescription medicines by visiting gout relief to avoid harmful side effects.

Article Source: http://EzineArticles.com/?expert=Rich_Rollend

Eliminating Gout Forever

Gout is a type of arthritis that is characterized by sudden burning pain, stiffness and swelling in a joint, often the big toe. The disease is hallmarked by elevated levels of uric acid in the bloodstream, caused by deposits of monosodium urate crystals which accumulate in the joints. It is normal for some uric acid to be present in the blood stream because the body is continually breaking down cells and forming new ones. The risk of gout is higher if the individual is overweight, drinks alcohol or eats food high in purines consistently. Familiar foods such as fish or meats can be high in purines and are precursors of uric acid. Purines are the colorless crystals which metabolize into uric acid, and when the kidneys cannot eliminate enough in the urine, the uric acid level becomes abnormally high.

The cause of gout is too much uric acid in the blood stream forming hard crystals inside the joints. It is a condition which affects men more than women and therefore eliminating gout forever is in their best interest.

Attacks of gout occur without warning often at night, accompanied by severe pain in one or more joints. The joint becomes swollen and the skin reddens and becomes extremely painful to the touch. The disorder most often affects the joint at the base of the big toe, but it can also commonly occur in the instep, ankle, knee, wrist and elbow. The first few attacks usually affect only one joint and can last a few days or many weeks before it is resolved. As the crystals dissolve from the joint area, the pain and discomfort will subside normally over a two week period. Some individuals may remain free from another flare-up for many months or years while others may never get another attack for the rest of their lives.

However, if the gout attack is not treated and the disorder progresses the affected joint can be permanently damaged. Severe, chronic gout will cause the joint to become stiff and have limited mobility. Eliminating gout forever is especially critical for those individuals suffering the chronic stage of this condition and experiencing the persistently painful joints, swelling and tenderness common with gout.

Gout is often diagnosed on its symptoms and joint examination. A high uric acid level in the blood will support the diagnosis as well as joint aspiration to pull out fluid in the joint affected. This fluid is sent to the lab and viewed under a polarized microscope. With early diagnosis and treatment and long term dietary changes in lifestyle choices, many individuals will never reach the chronic stage. The changes which are required in eliminating gout forever and to ensure that the chronic stage is never reached are minor compared to the suffering endured by these chronic gout symptoms.

The first step is to relieve the pain and to stop the acute inflammation. Sometimes the same needle used to remove fluid will be used to inject medication to effectively terminate the inflammation caused by urate crystals. The second step is to prevent further attacks. This involves maintaining adequate fluid intake, reducing weight, dietary changes and reduction in alcohol consumption. Long term treatment and prevention is essential for the health of the individual and in eliminating gout forever.

The author, John Donnelly, has spent many hours researching the problems associated with gout, and highly recommends the website below help in your quest for eliminating gout forever.



Article Source: http://EzineArticles.com/?expert=John_Donnelly

Tuesday, June 23, 2009

Value of Inflammation Marker in Patients with Intercritical Gout Arthritis

Writer : Endy Adnan
(supervised by Edu S Tehupeiory)

Background. Intercritical gout arthritis is an acute gout in intermittent period, caused by inflammation process neither with tofus. Inflammation is a complex mechanism among pathogenic agents, parenchymal cell, tissue and blood vessels either with plasma or blood cellular. Inflammation induced by uric crystal in the joint is a patomechanismin gout arthritis and than activated and release celluler, humoral or complement mediators. The aim of this study to investigatigate the value of leucocyte, ESR, CRP, C3 components and C4 components in intercritical gout arthritis.
Subject and Method. This is an observational study with case control approach conducted in 20 subjects with intercritical gout arthritis (case) and 20 with asymptomatic hyperuricemia (control). Eclusioc criteria were rheumatoid arthritis, septic arthritis, psoriatic arthritis, osteoarthritis, chronic infection disease, and history of trauma, diagnosed as anemia, chronic kidney disease, malignancies, during consumption diuretics or AINS.
Result. We found mean age 54,93 year old, uric acid 9,445 mg/dl, 5 with positif CRP. Mean value of leucocyte both case and control were 9988,25 +- 2819,936 / mm3 vs 9540,5 +- 2333,656 / mm3 (p=0,153). No correlation between intercritial gout arthritis and the higher value of leucocyte (p=0,11), (OR 1.625; Cl 0.869-3.038). Mean value of ESR were 58,70 +- 12,650 vs 36,75 +- 7,587212 (p=0.343) and the statistical correlation between value of C4 and intercrtitical gout arthritis was uncalculated because of invariant data.
Conclusion. The higher value of ESR and CRP were correlated in intercritical gout arthritis comparing with asymtomatic hyperuricemia, while leucocyte, C3 and C4 were not correlated.

Association Between C-Reactive Protein Level and Components of the Metabolic Syndrome

Writer : Daniel Josten
(supervised by John M.F. Adam)

Association between C-reactive protein, a sensitive marker for inlammation, and the development of atherosclerosis disease had been observed in experimental and epidemological studies. Interest in CRP was increased along with problem of metabolic syndrome because basically all components of metabolic syndrome were inflammation process. In Indonesia, the same study was so limited, eventhough according to WHO prevelance of obesity was increased in developing countries so do in developed countries wich will increase the metabolic syndrome population. The aim of this study is to know association between CRP level and components of metabolic syndrome. We studied 138 apparently health subjects aged 35-65 years old whoparticipated in the East Indonesia Diabetes Epidemiology Group (EIDEG) in November 2005 until January 2007. Method in this study was observational study with cross sectional approach. Anamnesis and complete physical examintaion was performed to all subjects included blood pressure and waist circumference. After 12-h fasting, blood was taken for fasting plasma glucose (FPG), HDL-cholesterol, triglyseride and C-reactive protein (CRP). For the diagnosis of Mets we used criteria defined by the International Diabetes Federation (IDF) 2005. The result of this study showed there were a statistically significant positive correlation between CRP and waist circumference (r=0.1), TG (r=0.3), FPG (r=0.2), blood pressure (r=0.3) (all p<0.05). A negative correlation was found between CRP and HDL cholesterol (r=0.3) (p=0.02). Mean CRP levelth 0 components of the metabolic syndrome were 1.04, 1.52, 3.15, 4.21, 4.65, and 6.33 mg/l respectively (r=0.54, p <0.0001). From this study we concluded that CRP leel were associated with components of metabolic syndrome.

Sunday, June 21, 2009

The Relation Between Components of Metabolic Syndrome with Plasminogen Activator Inhibitor-1

Writer :Himawan Dharmayani Sanusi

(supervised by John MF Adam, A. Fachruddin Benyamin)


Plasminogen activator inhibitor-1 is an acute phase protein that regulate fibrinolysis from hemostatic process and plays an important role in the pathogenesis of atherothrombosis. Subject with metabolic syndrome are high risk of cardiovascular complication. The aim of this study is to find out of relation between PAI-1 and the component of metabolic syondrome. In this study, we enrolled subject of age varied from 30 – 65 years. Those with diabetes mellitus, and clinical cardiovascular complications were excluded. History taking and complete physical examinations were performed to all subject. After 12 hour fasting,blood was taken for the examinations of fasting HDL –cholestrol ,triglyceride fasting flasma glucose and PAI -1.PAI -1 was measured by sensitive enzyme immunoassay technique for the diagnosis of metabolic syndrome, the AHA/NHLBI guideline was used with the modification of waist circumeference fro Asian population.Statistical analysis using SPSS 12 for windows.

During the study,183 subjects can be covered , 41% males and 59 % females. PAI – 1 levels was statistical significant higner among patients with metabolic syndrome compared to normal subjects,18.01 ± 9.33 mlU/ml and 4.33± 4.83 mlU/ml subsequently [ P= 0.000 ] . there was significant positive correlation between fasting hyperglycemia , triglycerides,waist circumference,blood pressure with increased of PAI – 1 and significant negatife correlation between HDL – chosterol with increased levels of PAI – 1 [p<> 7 IU /ml as a high level,statistical analysis showed that subjects with high trigly cerydes had the higner risk for hig PAI – 1 activity compared to correlation between the componenst of metabolic syndrome with the increasing of PAI – 1 levels, being the highest among those with five components .[r =0.828 and p=0.000]

In conclusion , there was a corelation between the components of metabolic syndrome and levels of plasminogen activator inhibitor – 1 those with hypetriglycerides had the highest risk to have high levels of plasminogen activator inhibitor – 1.